After absorption through the leaves and roots of susceptible species, Arylex molecules are metabolised to the active form and translocated symplastically throughout the vascular system accumulating at the plant’s growing points.
Arylex molecules are rapidly absorbed into the plant’s cells where the herbicide mimics naturally occurring hormones by binding with specific auxin receptors in the cell’s nucleus – it is this action of binding that differentiates Arylex from other synthetic auxin herbicides.
Binding of Arylex with these auxin receptors triggers the degradation of repressor proteins that normally stabilize auxin-regulated genes in the cell nucleus.
Without the repressor proteins in place, the auxin-regulated genes “switch-on,” inducing a cascade of unregulated growth in susceptible plants just minutes after application.
Within hours after application the uncontrolled growth disrupts multiple plant processes causing a loss of normal growth function.
In only a few hours to a few days, visual symptoms appear and continue to develop leading to death in susceptible species a few weeks after application.